Caloric restriction attenuates beta-amyloid neuropathology in a mouse model of Alzheimer's disease.

Diseases which may be prevented or cured by means of therapeutic fasting and caloric restriction

(experimental and clinical evidence: click to see scientific report)

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The Anti-Aging Fasting Program consists of a 7-28 days program (including three - fourteen fasting days) 7-10-14-18-28-day low-calorie

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Scientific update

Caloric restriction attenuates beta-amyloid neuropathology in a mouse model of Alzheimer's disease.

This study was designed to explore the possibility that caloric restriction (CR) may benefit Alzheimer's disease (AD) by preventing beta-amyloid (Abeta) neuropathology pivotal to the initiation and progression of the disease. We report that a CR dietary regimen prevents Abeta peptides generation and neuritic plaque deposition in the brain of a mouse model of AD neuropathology through mechanisms associated with promotion of anti-amyloidogenic alpha-secretase activity. Study findings support existing epidemiological evidence indicating that caloric intake may influence risk for AD and raises the possibility that CR may be used in preventative measures aimed at delaying the onset of AD amyloid neuropathology

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